The theory of hemodynamics is rife with uncertainty. The left ventricle cannot perform the amount of labour required to move the blood through the circulatory system. As a result, blood flows more slowly in between heartbeats, altering the pattern of the accompanying electrocardiogram's Womersley number (ECG). Coagulation exhibits strong viscoelastic change. The blood transient flow resistance and the ECG must be correlated. It was investigated how the electromagnetic field affected blood coagulation. In 25 healthy subjects, the oscillated electromagnetic field (500–5000 Hz) with the square wave input signal had an impact on venous blood (15 males, 10 females in the age 18 - 57 years). Time of the sample's electromagnetic irradiation (EMI): 3–10 min. In normal blood samples, hypocoagulation was seen together with thrombolysis following blood stasis (decreased platelet quantity up to 10–23 109/L, Prothrombin index up to 9%–10%, Fibrinogen concentration up to 0.20–0.21 g/L). Electroacoustic phenomena are started by the cardiac depolarization's ac electric field. Together with the heart's pulse pressure, a growing repulsive electromagnetic force operates on red blood cells (RBC) to drive blood mobility and viscoelastic changes. The magnetic properties of haemoglobin make it easier for changes to the blood's inertia, elasticity, and hemodynamics. The external electromagnetic signal has the ability to control all aspects of thrombolysis and blood coagulation.